Lei et al. show that HLS1 negatively regulates plant senescence and the ABS3-mediated senescence pathway by interacting with ATG8 and attenuating the ABS3-ATG8 interaction and the ability of ABS3 to promote senescence. Their study provides insights into the functional link between HLS1, ABS3, and ATG8 in plant senescence regulation.

Since the beginning of time, man has been interested in what happens after death.

Although there are numerous traditional answers to this question, it’s possible that scholars have added countless more ideas merely to provide some variation.

According to Robert Lanza, M.D., death is just a doorway to an endless number of universes. Furthermore, according to Lanza, everything that may possibly happen in our lifetime has already happened. He continues by saying that death does not exist in these situations because all of these possibilities are happening at the same time. We only connect our consciousness to our physical bodies because of the energy that flows through our brains.

A different kind of human cell division could improve our understanding of cancer and help us grow specific tissues

New research from the University of Bristol has uncovered striking links between immune system proteins and neuropsychiatric conditions, including schizophrenia, depression, and Alzheimer’s disease. By analyzing large genetic datasets using Mendelian randomisation, scientists identified 29 immune-related proteins potentially playing a causal role in these disorders.

The findings suggest that mental health conditions may not be isolated to the brain but involve the entire body, potentially reshaping future treatment strategies. This video explores how inflammation and immune pathways could be the next frontier in neuropsychiatric care.

#mentalhealth #immunesystem #neuroscience #health #psychology #depression

A massive brain study reveals that the roots of consciousness may lie in perception, not thought.

Increasingly stricter regulations on emissions from lean-burn engines, such as the Euro 7 standard, are approaching. This requires the development of catalytic materials that can reduce the toxic nitrogen oxides efficiently at low temperatures. Researchers at the Department of Physics at Chalmers University of Technology, together with industrial partner Umicore, now present a study showing how machine learning could help engines run cleaner.

Catalytic converters reduce the amount of toxic pollutants emitted into the air from a vehicle’s exhaust system. Stricter regulations on emissions standards within the coming years, such as the European Union’s proposed Euro 7, aim at further reducing air pollution from vehicles. Therefore, improved catalysts are needed to limit the emissions of harmful pollutants.

The main technology of selective catalytic reduction of nitrogen oxides uses ammonia as a reducing agent. Thus, the catalytic material should promote the formation of a nitrogen–nitrogen bond between nitrogen oxides and ammonia in an oxygen-rich environment and prevent unwanted reactions, which include the oxidation of ammonia to even more nitrogen oxides or nitrous oxide.

Scientists are racing against time to try and create revolutionary, sustainable energy sources (such as solid-state batteries) to combat climate change. However, this race is more like a marathon, as conventional approaches are trial-and-error in nature, typically focusing on testing individual materials and set pathways one by one.

To get us to the finish line faster, researchers at Tohoku University developed a data-driven AI framework that points out potential solid-state electrolyte (SSE) candidates that could be “the one” to create the ideal sustainable energy solution.

This model does not only select optimal candidates, but can also predict how the reaction will occur and why this candidate is a good choice—providing interesting insights into potential mechanisms and giving researchers a huge head start without even stepping foot into the lab.

In a large population-based cohort of individuals who underwent electron-beam CT, coronary artery calcium score was independently associated with incident lung cancer diagnosis but did not demonstrate potential to improve risk stratification in lung cancer screening.

Senolytics can prevent and treat age-related diseases in mice, but data in humans are lacking — for now.

The common cold sore virus, which is often caught in childhood, usually stays in the body for life—quietly dormant in the nerves. Now and then, things like stress, illness or injury can trigger it, bringing on a cold sore in some people. But this same virus—called herpes simplex virus type 1—may also play an important role in something far more serious: Alzheimer’s disease.

Over 30 years ago, my colleagues and I made a surprising discovery. We found that this cold sore virus can be present in the brains of older people. It was the first clear sign that a virus could be quietly living in the brain, which was long thought to be completely germ-free—protected by the so-called “blood-brain barrier.”

Then we discovered something even more striking. People who have a certain version of a gene (called APOE-e4) that increases their risk of Alzheimer’s, and who have been infected with this virus, have a risk that is many times greater.