DNA changes in umbilical cord blood may help predict which children are at higher risk for future health issues like diabetes, liver disease, and stroke.
Category: biotech/medical – Page 104
Brain-Computer Interface: No Open Brain Surgery Required đ§
Synchron has developed a Brain-Computer Interface that uses pre-existing technologies such as the stent and catheter to allow insertion into the brain without the need for open brain surgery.
Read the CNET article for more info:
You Might Not Need Open Brain Surgery to Get Mind Control https://cnet.co/3sZ7k67
0:00 Intro.
0:25 History of Brain Chip Implants.
0:44 About Synchron.
0:54 How Synchron implants the interface.
1:55 How brain patterns transmit signals.
2:50 Risks and Concerns.
3:50 Patients and Clinical Testing.
4:25 Brain Health Monitoring.
5:04 Synchron Switch Price.
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#WhatTheFuture #Synchron #BCI
Augmenting Your Brain with AI Chips â Crazy or Futuristic?
Improving the human brain with the help of implanted chips and AI: Elon Muskâs startup Neuralink is among the companies developing this tech. Initial tests show it could be a game changer for people with disabilities.
#technology #braincomputerinterface #bci #australia #medicine #optimization #elonmusk #neuralink #stentrode #brain #computer #dishbrain.
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Beyond the double helix: Alternative DNA conformations in ape genomes
Certain DNA sequences can form structures other than the canonical double helix. These alternative DNA conformationsâreferred to as non-B DNAâhave been implicated as regulators of cellular processes and of genome evolution, but their DNA tends to be repetitive, which until recently made reliably reading and assembling their sequences difficult.
Now, a team of researchers, led by Penn State biologists, has comprehensively predicted the location of non-B DNA structures in great apes. Itâs the first step in understanding the functions and evolution of such structures, known to contribute to genetic diseases and cancer, the team said.
The work depends on newly available telomere-to-telomere (T2T), or end-to-end, genomes of humans and other great apes that overcame sequencing and assembly difficulties associated with repetitive DNA to fill in any remaining gaps in the genomes. A paper describing the study, which shows that non-B DNA is enriched in the newly sequenced segments of the genomes and suggests potential new functions, was published in the journal Nucleic Acids Research.
Brain networks study examines the transition from early to chronic psychosis
Patients in the early stages of psychosis respond to treatments differently than those who have developed a chronic version of the disorder. Understanding the neurobiological changes from early to chronic stages is essential for developing targeted prevention and treatment strategies. But how symptoms change during this transitionâand what role the brain playsâis unclear.
Researchers at Yale School of Medicine (YSM) have now examined patients with early and chronic forms of psychosis to map symptom evolution and identify relevant brain networks. They published their findings in the journal Neuropsychopharmacology.
âWe are interested in how psychosis and psychiatric disorders develop,â says Maya Foster, first author of the study and a Ph.D. student in the lab of Dustin Scheinost, Ph.D., associate professor of radiology and biomedical imaging at YSM.
Psychosis-inducing lesions affect a common brain circuit in the hippocampus, research reveals
A new study investigated the brain circuits involved in psychosisâa condition characterized by delusions, hallucinations, disorganized thinking and detachment from reality.
Andrew Pines, MD, MA, a resident in the Department of Psychiatry at Brigham and Womenâs Hospital and a researcher in the Center for Brain Circuit Therapeutics, is the lead author of the paper published in JAMA Psychiatry titled âMapping Lesions That Cause Psychosis to a Human Brain Circuit and Proposed Stimulation Target.â
Psychosis is the classic symptom of schizophrenia, a serious mental illness that causes marked disability in otherwise young and healthy patients. The researchers analyzed published cases in which focal brain damage caused psychosis, with the idea that if damaging a brain circuit causes a symptom, then mapping that circuit might tell us about how to treat that symptom.
Emergence of psychosis in Alzheimer disease links to elevations in p-tau181
For individuals with Alzheimer disease (AD), the emergence of psychosis is associated with elevations in levels of plasma tau phosphorylated at threonine 181 (p-tau181), according to a study published online June 26 in JAMA Psychiatry.
Jesus J. Gomar, Ph.D., and Jeremy Koppel, M.D., from the Feinstein Institutes for Medical Research in Manhasset, New York, examined the longitudinal dynamics of p-tau181 and neurofilament light chain protein (NfL) levels in association with the emergence of psychotic symptoms. Patients with mild cognitive impairment (MCI) and AD (with psychosis [AD+P] and without psychosis [ADâP]) and participants who were cognitively unimpaired (CU) were compared at baseline.
For the longitudinal analysis, participants with MCI and AD were categorized into those with evidence of psychosis at baseline and those who showed incidence of psychosis over the course of the study. The cohort included 752 participants with AD and 424 CU participants.