Dr. Judith Campisi, a professor at the Buck Institute for Research on Aging, focuses her lecture on senescent cells and their role in cancer and aging. She explains how cancer is an age-related disease by describing the many conditions beyond DNA mutations that must generally be met for a malignant tumor to form. Dr. Campisi acknowledges that while cellular senescence is a powerful anti-cancer mechanism and while senescent cells may even play a key role in wound healing, senescent cells can nonetheless cause inflammation in their local environment and actually support the formation of tumors.
Last weekend, an invite-only group of about 150 experts convened privately at Harvard. Behind closed doors, they discussed the prospect of designing and building an entire human genome from scratch, using only a computer, a DNA synthesizer and raw materials.
The artificial genome would then be inserted into a living human cell to replace its natural DNA. The hope is that the cell “reboots,” changing its biological processes to operate based on instructions provided by the artificial DNA.
In other words, we may soon be looking at the first “artificial human cell.”
Presumably, this also will make further research on patients in this state harder, since they are potentially savable and can be harmed by some interventions.
Whose brain is it anyway?
The tougher, ethical question is whether this actually would help the deceased person, or (assuming it works) even bring about a new person.
Biography: Stuart Russell received his B.A. with first-class honours in physics from Oxford University in 1982 and his Ph.D. in computer science from Stanford in 1986. He then joined the faculty of the University of California at Berkeley, where he is Professor (and formerly Chair) of Electrical Engineering and Computer Sciences and holder of the Smith-Zadeh Chair in Engineering. He is also an Adjunct Professor of Neurological Surgery at UC San Francisco and Vice-Chair of the World Economic Forum’s Council on AI and Robotics. He has published over 150 papers on a wide range of topics in artificial intelligence including machine learning, probabilistic reasoning, knowledge representation, planning, real-time decision making, multitarget tracking, computer vision, computational physiology, and global seismic monitoring. His books include “The Use of Knowledge in Analogy and Induction”, “Do the Right Thing: Studies in Limited Rationality” (with Eric Wefald), and “Artificial Intelligence: A Modern Approach” (with Peter Norvig).
Abstract: Autonomous weapons systems select and engage targets without human intervention; they become lethal when those targets include humans. LAWS might include, for example, armed quadcopters that can search for and eliminate enemy combatants in a city, but do not include cruise missiles or remotely piloted drones for which humans make all targeting decisions. The artificial intelligence (AI) and robotics communities face an important ethical decision: whether to support or oppose the development of lethal autonomous weapons systems (LAWS).
While pregnant with her first child, Rhea Birusingh started experiencing blurry vision that her OB-GYN dismissed as an expected pregnancy-related change, but three months later, she went to her ophthalmologist, who discovered an inoperable 2-centimeter benign brain tumor behind her right eye. Now, nearly four months later, Birusingh’s son is healthy and her vision is normal, thanks to a powerful, precise radiation treatment.
“When you’re a pathologist and your eyes are a money maker, you start to get a little bit worried,” Birusingh, 37, of Howey-in-the-Hills, Florida, told FoxNews.com.
A specific type of immune cell, called microglia (green) can help contain amyloid plaques (magenta), the key hallmark of Alzheimer’s disease, thus limiting their damage to surrounding brain cells.
But, Dr Jaime Grutzendler, associate professor of neurology and neuroscience at Yale, said that is no longer thought to be the case, and should signal a new.
He said: ‘It suggests we should be enhancing the function of these immune cells, not trying to suppress it.’
Harvard you’re wrong. Why? Harvard needs to explain to all the families with genetic mutations tied to various types of cancer, cancer survivors who carry a genetic mutation, or the workers who were exposed at work to radiation or carcinogens how their smoking caused their cancer when they never smoked in their entire life. However, how do you explain that to the countless million patients, survivors, and loved ones who lost someone to cancer that never smoked or were exposed to smoking.
As many as 40 percent of cancer cases, and half of cancer deaths, come down to things people could easily change, researchers said Thursday.
While Americans often worry about whether chemicals, pollution or other factors out of their control cause cancer, the new analysis shows otherwise: People are firmly in charge of much of their own risk of cancer.
Biologists have identified the one gene that caused the evolution of single-celled organisms into multicellularity, debunking previous theories that several genes were at play. The gene retinoblastoma is also the same gene that is found to be defective in cancer patients, and suppresses tumors.
