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New digital light manufacturing approach resolves common problems associated with 3D printing

A team of materials scientists, medical researchers and engineers affiliated with a large number of institutions across Australia has developed a new way to conduct digital light manufacturing that overcomes problems with current methods. In their paper published in the journal Nature, the group describes their new technique, how it works and ways it might be used.

Study Identifies Neural Crest Stem Cells as Reprogramming Drivers

Summary: Neural crest stem cells, a rare type found in skin and other tissues, are uniquely capable of reprogramming into different cell types, challenging the prevailing belief that any mature cell can be reprogrammed. The study reveals that cellular reprogramming is likely limited to these specialized stem cells rather than all mature cells.

Neural crest stem cells are present in skin, bone, and connective tissue, with a natural predisposition for transformation due to their origin in embryonic development. This finding could reshape strategies for stem cell therapies, emphasizing the role of neural crest cells in treating neurodegenerative diseases. The team hopes their work will refine cell reprogramming approaches and inspire further research into the specific potentials of stem cell types.

Exosomes Break Rat Lifespan Record

This has to do with E5, but last I checked they were only doing skin rejuvenation treatments in people.


In Aging Cell, researchers have published their findings that exosomes, which we have previously reported to extend the lives of mice, also extend the lives of rats.

Known to be effective

Exosomes, a subset of extracellular vesicles (EVs), can be visualized as messages and packages that cells send to one another. Along with lifespan studies, EVs have been investigated for their ability to treat liver fibrosis, and they have been identified as potential biomarkers of disease [1].

Scientists successfully reverse liver fibrosis in mice

Cirrhosis, hepatitis infection and other causes can trigger liver fibrosis—a potentially lethal stiffening of tissue that, once begun, is irreversible. For many patients, a liver transplant is their only hope. However, research at Cedars-Sinai in Los Angeles may offer patients a glimmer of hope. Scientists there say they’ve successfully reversed liver fibrosis in mice.

Reporting in the journal Nature Communications, the team say they’ve discovered a genetic pathway that, if blocked, might bring fibrosis to a halt.

The three genes involved in this fibrotic process are called FOXM1, MAT2A and MAT2B.

Team engineers New Enzyme to Produce Synthetic Genetic Material

Discovery advances development of new therapeutic options for cancer and other diseases. A research team led by the University of California, Irvine has engineered an efficient new enzyme that can produce a synthetic genetic material called threose nucleic acid. The ability to synthesize artificial chains of TNA, which is inherently more stable than DNA, advances the discovery of potentially more powerful, precise therapeutic options to treat cancer and autoimmune, metabolic and infectious diseases.

A paper recently published in Nature Catalysis describes how the team created an enzyme called 10–92 that achieves faithful and fast TNA synthesis, overcoming key challenges in previous enzyme design strategies.

Inching ever closer to the capability of natural DNA synthesis, the 10–92 TNA polymerase facilitates the development of future TNA drugs.

Stem cell-like approach in plants sheds light on specialized cell wall formation

A new method developed by Penn State biologists allows them to turn stripped-down plant cells into other types of cells, similar to the way stem cells differentiate into different cell types. Using this method, the research team explored the banding patterns that increase the stability of plant cell walls—much like the corrugated patterns in cardboard—and how they are created. Additionally, the researchers revealed how the assembly of these structures can go astray in different mutant plant cells, which they said could ultimately inform methods to break down plant cells for biofuels.

Introducing The First SYNTHETIC AI HUMAN With Real Muscles (FAKE HUMANS SOON)

EngineAI’s SE01 humanoid robot redefines robotics with its smooth, human-like movement powered by advanced AI neural networks, showcasing a new level of realism in robotic technology. Clone Robotics pushes the boundaries further, creating a lifelike torso with synthetic muscles and joints that replicate the human musculoskeletal system, setting a new standard in AI-driven, realistic robotics. These innovations from EngineAI and Clone Robotics are transforming the future of humanoid robots, bringing AI and robotics closer to lifelike androids capable of human-like behavior, movement, and dexterity.

🔍 Key Topics Covered:
EngineAI’s groundbreaking humanoid robot, SE01, with AI-driven natural movement that mimics human gait.
Clone Robotics’ advanced torso robot, featuring synthetic muscles and joints for lifelike movement.
Real-world applications and implications for humanoid robots in industries, education, and daily life.

🎥 What You’ll Learn:
How EngineAI achieved smooth, human-like movement in SE01 through a unique neural network approach.
Clone Robotics’ development of a lifelike torso that mirrors the human musculoskeletal structure.
The future of humanoid robots as they move beyond warehouses, with potential roles in schools, hospitals, and even homes.

📊 Why This Matters:
This video dives into the latest advancements in humanoid robotics, revealing how companies like EngineAI and Clone Robotics are pushing the limits of AI and robotics. As robots grow more human-like, their potential to revolutionize industries and daily life becomes increasingly real.

DISCLAIMER:
This video explores EngineAI’s SE01 and Clone Robotics’ torso robot, highlighting their remarkable strides in lifelike robotics. Anyone curious about AI, robotics, and the future of humanoid technology will find this information insightful.

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