Lifeboat Foundation

A tissue-based soft robot that mimics the biomechanics of a stingray has been developed, which could lead to advances in bio-inspired robotics, regenerative medicine and medical diagnostics.

The simple body design of stingrays, specifically, a flattened body shape and side fins that start at the head and end at the base of their tail, makes them ideal to model bio-electromechanical systems on.

The 10-millimeter long robot is made up of four layers: tissue composed of live heart cells, two distinct types of specialized biomaterials for structural support, and flexible electrodes. Imitating nature, the robotic stingray is even able to “flap” its fins when the electrodes contract the heart cells on the biomaterial scaffold.

Continue reading “Bio-inspired robots come closer to reality” »

The famed startup incubator Y Combinator put out a call for companies that want to increase human longevity and “health span.”

Who they want: Founders with new ideas for treating old-age diseases like Alzheimer’s, “but we will also consider more radical anti-aging schemes,” YC president Sam Altman told MIT Technology Review.

Why longevity? Efforts to stop old age don’t actually get funded much. “My sense is that economic incentives of drugs companies are screwed up” says Altman. “I don’t think we have enough people saying, How can we make a lot of people a lot heathier?”

Continue reading “Y Combinator will give you $1 million to try to cure aging” »

After losing her faith, a former evangelical Christian felt adrift in the world. She then found solace in a radical technological philosophy – but its promises of immortality and spiritual transcendence soon seemed unsettlingly familiar by Meghan O’Gieblyn.

https://www.facebook.com/letonya.moore.5/videos/1970919586269818/

Researchers have found that by manipulating a single RNA molecule, they can reverse some aspects of cellular aging and regenerate aged cells.

Old cells resist regeneration

As we grow older, our cells gradually age, leading to the development of various diseases. Therefore, inducing cellular regeneration is one of the approaches that researchers are using to combat the age-related diseases associated with cellular aging. Unfortunately, aged cells are often highly resistant to therapies aimed at inducing regeneration.

Continue reading “Zeb2-NAT Molecule May Reverse Cellular Aging” »

Early Bird ends soon!

Have you got your tickets yet? If not, then you just have a few days to do so and save. Early Bird pricing ends on Monday, 11:59 pm CET.

The 2018 Undoing Aging Conference is focused on the cellular and molecular repair of age-related damage as the basis of therapies to bring aging under full medical control.

Continue reading “The 2018 Undoing Aging Conference” »

J147 is an experimental drug that has been shown to treat Alzheimer’s disease, and it also appears to reverse some aspects of aging. It is also poised to enter human clinical trials in the near future, although how it works has been somewhat of a puzzle.

A new study published in the journal Aging Cell has changed all that, and the results are quite intriguing[1]. Researchers at the Salk Institute have solved the mystery of how J147 works and why it makes old flies, mice, and cells more youthful.

We have all heard that exercise is good for our health. However, it can not only keep you healthy, it can also slow down some aspects of aging. Some researchers even think that it might be possible to use this knowledge to develop new therapies against aging. While waiting for that to happen, we need to exercise in order to slow down the effects of aging.

How important is it to keep fit?

So, how beneficial is exercising? Well, one of the best studies conducted on this subject showed that women will live 5.6 years longer and men 6.2 years longer if they exercise between 1 and 2.5 hours per week[1]. This makes exercise a better lifestyle choice than any other, at least as long as you’re not counting avoiding downright dangerous behavior, such as smoking.

https://www.youtube.com/watch?v=dsJiZbLPqNQ